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Host
0
Species Reactivity
0
Species Reactivity
0
Conjugate
0
Conjugate
0
Compatible Species
0
Conjugate
0

验证数据展示

  • WB analysis using 82959-1-RR (same clone as 82959-1-PBS)

    WB analysis using 82959-1-RR (same clone as 82959-1-PBS)

    Various lysates were subjected to SDS PAGE followed by western blot with 82959-1-RR (SCN3B antibody) at dilution of 1:5000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 82959-1-PBS in a different storage buffer formulation.

  • Affinity and Kinetic Characterization of 82959-1-PBS

    Affinity and Kinetic Characterization of 82959-1-PBS

    Biolayer interferometry (BLl) kinetic assays of 82959-1-RR against Human SCN3B were performed. The affinity constant is 0.429 nM.

SCN3B Recombinant antibody, PBS Only

SCN3B Uni-rAbTM Recombinant Antibody for WB, Indirect ELISA
Cat No. 82959-1-PBS

产品说明书

CloneNo. 230316F8

宿主/亚型

Rabbit / IgG

种属反应性

Human, rat, mouse

应用

WB, Indirect ELISA

别名

Sodium channel subunit beta 3, Sodium channel regulatory subunit beta-3, SCNB3, KIAA1158, HSA243396

缓冲液配方:  PBS Only
PBS and Azide
PBS Only
偶联物:  Unconjugated
规格: 

-/ -

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产品信息

82959-1-PBS targets SCN3B in WB, Indirect ELISA applications and shows reactivity with Human, rat, mouse samples.

经测试应用 WB, Indirect ELISA Application Description
经测试反应性 Human, rat, mouse
免疫原 SCN3B fusion protein Ag23201 种属同源性预测
宿主/亚型 Rabbit / IgG
抗体类别 Recombinant
产品类型 Antibody
全称 sodium channel, voltage-gated, type III, beta
别名 Sodium channel subunit beta 3, Sodium channel regulatory subunit beta-3, SCNB3, KIAA1158, HSA243396
观测分子量29 kDa
GenBank蛋白编号BC126265
基因名称 SCN3B
Gene ID (NCBI) 55800
偶联类型 Unconjugated
形式 Liquid
纯化方式Protein A purification
UNIPROT IDQ9NY72
储存缓冲液 PBS only , pH 7.3
储存条件Store at -80°C.
The product is shipped with ice packs. Upon receipt, store it immediately at -80°C

背景介绍

The sodium voltage-gated channel beta subunit 3 (SCN3B) plays a crucial role in electrically excitable cells and conduction tissue in the heart. Some previous studies have established that genetic modification in sodium voltage-channel genes encoding for the cardiac β-subunits, such as SCN1B, SCN2B, SCN3B and SCN4B, can result in atrial fibrillation (AF). (PMID: 36362949)