验证数据展示
经过测试的应用
Positive FC detected in | human blood |
推荐稀释比
应用 | 推荐稀释比 |
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This reagent has been pre-titrated and tested for flow cytometric analysis. The suggested use of this reagent is 10 μl per 10^6 cells in a 100 µl suspension or 10 μl per 100 µl of whole blood. | |
Sample-dependent, Check data in validation data gallery. |
发表文章中的应用
IF | See 1 publications below |
FC | See 3 publications below |
产品信息
PE-65184 targets STRO-1 in IF, FC applications and shows reactivity with Human samples.
经测试应用 | FC Application Description |
文献引用应用 | IF, FC |
经测试反应性 | Human |
文献引用反应性 | human |
免疫原 | Human CD34 positive bone marrow cells 种属同源性预测 |
宿主/亚型 | Mouse / IgM, lambda |
抗体类别 | Monoclonal |
产品类型 | Antibody |
全称 | STRO-1 |
别名 | STRO-1 |
基因名称 | |
Gene ID (NCBI) | |
RRID | AB_2883954 |
偶联类型 | PE Fluorescent Dye |
最大激发/发射波长 | 496 nm, 565 nm / 578 nm |
形式 | Liquid |
纯化方式 | The purified antibody is conjugated with R-phycoerythrin (PE) under optimum conditions. The conjugate is purified by size-exclusion chromatography. |
储存缓冲液 | PBS with 0.09% sodium azide and 0.5% BSA. |
储存条件 | Store at 2-8°C. Avoid exposure to light. Stable for one year after shipment. |
实验方案
Product Specific Protocols | |
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FC protocol for PE STRO-1 antibody PE-65184 | Download protocol |
Standard Protocols | |
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Click here to view our Standard Protocols |
发表文章
Species | Application | Title |
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Front Cell Dev Biol Nrf2 Activation Is Involved in Cyclic Mechanical Stress-Stimulated Osteogenic Differentiation in Periodontal Ligament Stem Cells via PI3K/Akt Signaling and HO1-SOD2 Interaction. | ||
Stem Cell Res Ther Discovery and characterization of heterogeneous and multipotent fibroblast populations isolated from excised cleft lip tissue | ||
J Periodontol Sulfonated chitosan oligosaccharide alleviates the inhibitory effect of basic fibroblast growth factor on osteogenic differentiation of human periodontal ligament stem cells. | ||
J Dent Sci N-acetylcysteine promotes cyclic mechanical stress-induced osteogenic differentiation of periodontal ligament stem cells by down-regulating Nrf2 expression. |